Inflammation, Pregnancy and Hypertension

Nonfunded Featured Topic — Thursday, April 29, 2021 — 4:00 PM - 5:30 PM — Virtual Session, Room APS-11
Water and Electrolyte Homeostasis Section — Chair: Ananth Karumanchi — Co-Chair:

Speakers

• B Cell Depletion During Pregnancy Improves Hypertension, Natural Killer Cell Activation, and May Not Worsen Fetal Outcomes in Response to Placental Ischemia
  Nathan Campbell — Pharmacology and Toxicology, University of Mississippi Medical Center
  4:00 PM - 4:15 PM


• Neutralization of Natural Killer Cell Associated Cytokines Improves Vascular Function and Reduces Blood Pressure in Placental Ischemic Rats
  Olivia K. Travis — Pharmacology and Toxicology, University of Mississippi Medical Center
  4:15 PM - 4:30 PM


• Decreased Systemic Complement Activation Product C3a is Associated with a Reduction in Pancreatic β Cell Area in Islets of Female Rat Offspring following Chronic Placental Ischemia-induced Hypertension
  Jean F. Regal — Biomedical Sciences, University of Minnesota Medical School
  4:30 PM - 4:45 PM


• The Importance of B Cells in Causing Hypertension During Pregnancy; To B or Not To B
  Owen Thomas Herrock — Pharmacology and Toxicology, University of Mississippi Medical Center
  4:45 PM - 5:00 PM


• IL-17 Causes Hypertension and Multi-Organ Tissue Dysfunction which is Attenuated with Blockade of Agonistic Autoantibodies to the Angiotensin II Type I (AT1-AA) Receptor during Pregnancy
  Sarah Fitzgerald — Pharmacology and Toxicology, University of Mississippi Medical Center
  5:00 PM - 5:15 PM


• NLRP3 Plays a Causative Role in Vascular and Renal Dysfunction and Hypertension in Placental Ischemic Pregnancy
  Denise C. Cornelius — Emergency Medicine, University of Mississippi Medical Center
  5:15 PM - 5:30 PM