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Common Pathways of Angiotensin on Cardiovascular and Respiratory Responses Induced by High-Fat-Diet, Hypoxia and Renal Hypertension

Symposium — Sunday, April 7, 2019 — 3:30 PM - 5:00 PM — Convention Center, Room W312A
Pan-American Societies (SFiB) — Chair: Eduardo Colombari — Co-Chair: Rodrigo Del Rio
Cosponsored by AJP - Heart and Circulatory Physiology

Obesity and hypertension are the leading cause of death in the world. Obesity and overweight contribute to elevated blood pressure in many patients with essential hypertension. Experimental and clinical studies indicate that visceral obesity induces hypertension by increasing renal sodium reabsorption through multiple mechanisms including activation of the sympathetic nervous system (SNS). Obesity-related hypertension is associated with altered responsiveness of SNS and the renin-angiotensin-system (RAS), possibly leading to blunted sympatho-inhibitory and vasodilator effects in the renal and splanchnic vasodilator beds. The aims of this symposium are to discuss the common pathways of angiotensin on cardiovascular and respiratory responses induced by high-fat-diet, hypoxia, renal hypertension, and heart failure. The neural mechanisms that process such neuroautonomic information associates to the cardiovascular and respiratory adjustments during the development of hypertension and/or obesity and/or heart failure have been studied in different experimental models. The SNS and RAS are the common mechanism among those pathophysiologies; indeed, this symposium will discuss how those pathways might interact

Speakers

  • Cardiovascular and Respiratory Changes in High-Fat Diet Fed Adult Rats and in Offspring of High-Fat Diet Fed Dams
    Débora S.A. Colombari — Department of Physiology & Pathology, School of Dentistry of Araraquara - UNESP

  • Angiotensin II-Induced Endoplasmic Reticulum Stress in the RVLM Contributes to Cardiorespiratory Impairment in High-Output Heart Failure
    Rodrigo Del Rio — Av Libertador Bernardo O'Higgins, 340, P. Universidad Católica de Chile

  • Renovascular Hypertension Elevates Pulmonary Ventilation in Rats by Carotid Body-Dependent Mechanisms
    Eduardo Colombari — Department of Physiology & Pathology, School of Dentistry of Araraquara - UNESP





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